The SlLRR-RLK94-SlPti1a Module Enhances Salt Tolerance by Modulating Reactive Oxygen Species and Hormone Homoeostasis in Tomato.

SlLRR-RLK94 and SlPti1a form a plasma membrane complex that positively regulates salt tolerance in tomato, and disrupting it causes severe chlorosis and restricted root growth under salinity.

• Why it matters: Soil salinization limits tomato productivity, but how LRR-RLK signaling controls salt tolerance through coordinated ionic, oxidative, and hormonal balance is unclear.
• What they did: Using CRISPR/Cas9 knockout of SlLRR-RLK94 and VIGS silencing of SlPti1a, the study measured ROS and antioxidant responses, Na+/K+ ratios, and performed transcriptomic profiling under salt stress.
• The result: Loss of the module drives excessive reactive oxygen species, alters antioxidant enzyme activities, elevates Na+/K+ via uncontrolled Na+ buildup, and links SlLRR-RLK94 loss to photosystem disruption, diterpenoid pathway suppression, and gibberellin deficiency with abscisic acid accumulation.

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