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The SCHENGEN pathway regulates lignin barrier formation via the receptor-like cytoplasmic kinase BIK1 in plant immunity.
Nature Plants · · Journal Article
Jang, Jeon + more
Abstract ↗AI summary
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In Arabidopsis thaliana effector-triggered immunity, SGN3 (GSO1) activates BIK1 to control lignin barrier formation, linked to disease resistance.
- Why it matters: Lignin deposition is a key immune barrier, but the signalling route from immune perception to lignification was unclear.
- What they did: The study shows SGN3 (GSO1) and CIF peptides act in leaves, with SGN3 preferentially using BAK1 and directly activating BIK1; NLR-driven CIF–SGN3–BIK1 cooperates with pattern recognition receptors.
- The result: CIF–SGN3–BIK1 signalling promotes reactive oxygen species production and lignin polymerization during effector-triggered immunity, enabling confinement of pathogen spread and supporting disease resistance.
The findingWhy it mattersWhat they didThe result
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