Dietary PFOA exposure worsens colitis severity by increasing inflammation, epithelial damage, and microbiome disruption in a mouse model.
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Dietary PFOA exposure worsens colitis severity by increasing inflammation, epithelial damage, and microbiome disruption in a mouse model.
Decabromodiphenyl ethane (DBDPE) promotes cancer-like traits in human breast cells and zebrafish xenografts, indicating potential health risks of this flame retardant.
Aqueous PFOS exposure disrupts the link between gaseous carbon loss and mineral-associated carbon retention in wetland mesocosms, with effects varying by concentration.
Genetically predicted higher PFOA and PFOS levels are linked to specific kidney disease risks, with PFOA associated with hypertensive nephropathy and calculus of kidney.
PFAS induce widespread hormetic responses across diverse ecological models, with biphasic dose-response patterns observed from bacteria to vertebrates.
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Moving areas, week to 3 Oct 2026