The abstract is read at the publisher; the summary is JClub's.
In young pear fruits, a ROS-responsive PbZAT11/PbZAT12–PbLAC1 module promotes lignin polymerization and stone cell differentiation, while PbPUB8 suppresses it by degrading PbZAT11/12.
- Why it matters: Stone cell abundance and aggregation shape pear fruit quality, but the pathway linking ROS signals to the lignin polymerization machinery during stone cell formation is unclear.
- What they did: Using spatial patterns of stone cells, ROS levels, and PCD events, the study combined gene co-expression network analysis with biochemical assays to test PbZAT11/PbZAT12 regulation of PbLAC1 and PbPUB8 effects on PbZAT11/12 stability.
- The result: ROS enhances PbZAT11 and PbZAT12 expression; PbZAT11 and PbZAT12 directly activate PbLAC1 to promote lignin polymerization, whereas PbPUB8 mediates ubiquitination and degradation of PbZAT11/12, negatively regulating stone cell formation.
The findingWhy it mattersWhat they didThe result
AI summary of the abstract · may contain errors · not medical advice · read the paper.