The abstract is read at the publisher; the summary is JClub's.
Suppressing CsUGT84A22 in tea plants (Camellia sinensis) lowers sinapoyl-β-D-glucose (S-G) accumulation and weakens gray blight resistance during pathogen infection.
- Why it matters: UGTs shape stress-responsive glycoside biosynthesis, but the regulatory logic linking pathogen cues to specific UGT loci and antifungal glycosides is unclear.
- What they did: The study characterized a pathogen-induced UGT genomic locus activated by gray blight, tested CsUGT84A22 function in converting sinapic acid to S-G, and used gene suppression plus transcription-factor and JA-pathway perturbations.
- The result: CsMYB73 within the locus negatively regulated CsUGT84A22, while suppressing CsMYB73 increased S-G and gray blight resistance; JA biosynthesis and CsJAZ14-linked repression further modulated this pathway.
The findingWhy it mattersWhat they didThe result
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