The abstract is read at the publisher; the summary is JClub's.
Disrupting the ZmJAZ14–ZmMYC2a/b jasmonic acid module in maize prolongs the anthesis–silking interval (ASI), indicating it coordinates tassel–ear synchrony.
- Why it matters: Yield gains from trait evolution can impose physiological constraints, and it is unclear whether JA signaling coordinates ear and tassel development to prevent ASI extension.
- What they did: The study links ZmJAZ14–ZmMYC2a/b to reproductive-transition and development genes (SPL29, BA1, EXPA5) and tests effects of ZmMYC2a/b disruption or ZmJAZ14 accumulation on ASI.
- The result: ZmJAZ14–ZmMYC2a/b regulation supports synchronous growth of tassels and ears, while its disruption or repressor buildup prolongs ASI, implying a lineage-specific evolutionary burden.
The findingWhy it mattersWhat they didThe result
AI summary of the abstract · may contain errors · not medical advice · read the paper.