The abstract is read at the publisher; the summary is JClub's.
In Arabidopsis thaliana under inorganic phosphate deficiency, PEPR1/PEPR2 signaling is selectively sensitized, with Pep-triggered responses strongly potentiated while FLS2- and CERK1-linked pathways decline.
- Why it matters: Plants must reprogram nutrient acquisition during phosphate starvation without losing immune competence, but how PRR signaling is tuned by nutritional status is unclear.
- What they did: Using membrane proteomics and immunoblotting, the study measured PEPR1/PEPR2 abundance changes under Pi limitation and tested Pep-, flagellin-, and chitin-triggered responses; it also required LPR1/LPR2, RBOHD/RBOHF, and WRKY33.
- The result: PEPR-dependent signaling promotes both basal PSR transcriptional reprogramming and growth under Pi stress, and PEPR1/PEPR2 loss compromises bacterial resistance and shifts root-associated microbiota assembly.
The findingWhy it mattersWhat they didThe result
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