Tau filaments from human Alzheimer's and corticobasal degeneration brains propagate in mice, maintaining their distinct structures and supporting prion-like transmission.
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Prion Diseases and Protein Misfolding
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Multiple prion conformers coexist within single natural scrapie isolates, with at least one previously unrecognized conformer identified through enhanced detection methods.
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Protein misfolding enteropathy detected in GI biopsies predicts neurodegenerative disease with over 80% sensitivity up to 7 years before symptoms.
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Prion-based protein self-assembly creates heritable mutagenesis switches that accelerate adaptation in yeast and pathogenic fungi, with up to 300 million years of divergence.
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